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This azobenzene-based design allowed reverse photoregulation of NAMPT activity and downstream NAD + levels, crucially avoiding the off-target toxicity in the dark state that plagues inhibitor-based PS-PROTACs and thereby laying a foundation for clinical applications

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Current research focuses on cellular and animal models, while human studies on Se-enriched foods or compounds for AD ferroptosis prevention remain limited and warrant further exploration

doi:10.1146/annurev.cellbio.22.010605.093503